983 resultados para Nombres impairs
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Vermetids form reefs in sub-tropical and warm-temperate waters that protect coasts from erosion, regulate sediment transport and accumulation, serve as carbon sinks and provide habitat for other species. The gastropods that form these reefs brood encapsulated larvae; they are threatened by rapid environmental changes since their ability to disperse is very limited. We used transplant experiments along a natural CO2 gradient to assess ocean acidification effects on the reef-building gastropod Dendropoma petraeum. We found that although D. petraeum were able to reproduce and brood at elevated levels of CO2, recruitment success was adversely affected. Long-term exposure to acidified conditions predicted for the year 2100 and beyond caused shell dissolution and a significant increase in shell Mg content. Unless CO2 emissions are reduced and conservation measures taken, our results suggest these reefs are in danger of extinction within this century, with significant ecological and socioeconomic ramifications for coastal systems.
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Segunda obra con port. propia, en p. 133
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Sign.: A-H8, I-K4, L-P8
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Sign.: A-H8, I-K4, L-P8
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Incipit : "Prologo. Guillermo Febrer, nobilissimo docto y experimentado fue muy estimado de D. Jaime I de Aragon"
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Sign.: []4
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Hay un ejemplar encuadernado con: Discret rahonament, quiexa formal que fan contra el Micalet de la Seu, la Torre de Espioca, y la Torre de Paterna, sobre la gran visita que éste tingué en lo dia cinc de Deembre [sic] ... per veure y admirar tan magnífica obra y deliciosa vista ... Carlos Quart (que Deu guart) y el señor Don Fernando de Borbó ... : (XVIII/1105).
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Hay un ejemplar encuadernado con: Discret rahonament, quiexa formal que fan contra el Micalet de la Seu, la Torre de Espioca, y la Torre de Paterna, sobre la gran visita que éste tingué en lo dia cinc de Deembre [sic] ... per veure y admirar tan magnífica obra y deliciosa vista ... Carlos Quart (que Deu guart) y el señor Don Fernando de Borbó ... : (XVIII/1105).
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Experimental autoimmune encephalomyelitis (EAE) is a T cell autoimmune disorder that is a widely used animal model for multiple sclerosis (MS) and, as in MS, clinical signs of EAE are associated with blood–brain barrier (BBB) disruption. SR 57746A, a nonpeptide drug without classical immunosuppressive properties, efficiently protected the BBB and impaired intrathecal IgG synthesis (two conventional markers of MS exacerbation) and consequently suppressed EAE clinical signs. This compound inhibited EAE-induced spinal cord mononuclear cell invasion and normalized tumor necrosis factor α and IFN-γ mRNA expression within the spinal cord. These data suggested that pharmacological intervention aimed at inhibiting proinflammatory cytokine expression within the central nervous system provided protection against BBB disruption, the first clinical sign of EAE and probably the key point of acute MS attacks. This finding could lead to the development of a new class of compounds for oral therapy of MS, as a supplement to immunosuppressive agents.