Scribonii Largi Conpositiones [sic] /

Includes index.

Honzō wamyō.

Mode of access: Internet.

[Papers]

Binder's title.

Yamato honzō.

Cover title.

Ricerche Sperimentali

Mode of access: Internet.

Treatise on therapeutics,

Mode of access: Internet.

New and nonofficial drugs.

Descriptions of therapeutic, prophylactic and diagnostic agents evaluated by the Council on Drugs.

An analytical compendium of the various branches of medical science, for the use and examination of students,

pt. 1. A hand-book of anatomy.--pt. 2. A hand-book of physiology.--pt. 3. A hand-book of surgery.--pt. 4. A hand-book of obstetrics.--pt. 5. A hand-book of materia medica and therapeutics.--pt. 6. A hand-book of chemistry.--pt. 7. A hand-book of the practice of medicine.

Inhibition of epidermal growth factor receptor expression by RNA interference in A549 cells

AIM: To investigate the biological features of A549 cells in which epidermal growth factor (EGF) receptors expression were suppressed by RNA interference (RNAi). METHODS: A549 cells were transfected using short small interfering RNAs (siRNAs) formulated with Lipofectamine 2000. The EGF receptor numbers were determined by Western blotting and flowcytometry. The antiproliferative effects of sequence specific double stranded RNA (dsRNA) were assessed using cell count, colony assay and scratch assay. The chemosensitivity of transfected cells to cisplatin was measured by MTT. RESULTS: Sequence specific dsRNA-EGFR down-regulated EGF receptor expression dramatically. Compared with the control group, dsRNA-EGFR reduced the cell number by 85.0 %, decreased the colonies by 63.3 %, inhibited the migration by 87.2 %, and increased the sensitivity of A549 to cisplatin by four-fold. CONCLUSION: Sequence specific dsRNA-EGFR were capable of suppressing EGF receptor expression, hence significantly inhibiting cellular proliferation and motility, and enhancing chemosensitivity of A549 cells to cisplatin. The successful application of dsRNA-EGFR for inhibition of proliferation in EGF receptor overexpressing cells can help extend the list of available therapeutic modalities in the treatment of non-small-cell lung carcinoma (NSCLC).

Methods and processes for development of a CONSORT extension for reporting pilot randomized controlled trials

Peer reviewed

Pharmacovigilance in children : detecting adverse drug reactions in routine electronic healthcare records. A systematic review

This article is protected by copyright. All rights reserved.

Pharmacovigilance in children : detecting adverse drug reactions in routine electronic healthcare records. A systematic review

This article is protected by copyright. All rights reserved.

Commiphora gileadensis sap extract induces cell cycle-dependent death in immortalized keratinocytes and human dermoid carcinoma

We would like to dedicate this article to the memory of Professor Yoram Milner, who has passed away before this study was achieved. We are immeasurably indebted to him, professionally and personally. The study was supported by grants from the ICA Foundation and the Ministry of Science, Technology and Space.

The 9th European Conference on Marine Natural Products

Acknowledgments This work was supported by grants from the European Commission within its FP7 Programme, under the thematic area KBBE.2012.3.2-01 with Grant Number Nos. 311932 “SeaBioTech”, 311848 “BlueGenics”, and 312184 PharmaSea.

International expert opinion on the management of infection caused by azole-resistant Aspergillus fumigatus

Acknowledgements We thank Gilead Sciences Europe Ltd., UK for financially supporting logistical aspects of this study. The logistics of the meeting were handled by Congress Care, Den Bosch, The Netherlands. The sponsor was not involved in the selection of the participants or procedures, or in the discussion, data collection, analysis or writing of the manuscript. The medical writer was financially supported by the Dutch Society for Medical Mycology and the Department of Medical Microbiology, Radboud University Medical Centre, Nijmegen, The Netherlands.