A geographical sketch of that part of North America, called Oregon: containing an account of the Indian title;--the nature of a right of sovereignty;--the first discoveries;--climate and seasons;--face of the country and mountains--natural divisions, physical appearance and soil of each;--forests and vegetable productions;--rivers, bays; &c.; islands, &c.; animals;--the disposition of the Indians, and the number and situation of their tribes;--together with an essay on the advantages resulting from a settlement of the territory. To which is attached a new map of the country.

Mode of access: Internet.

Health [a Monthly Devoted to the Cause and Cure of Disease]

Mode of access: Internet.

Health; Devoted to the Cause and Cure of Disease

Title Varies: Mar.1916-Mar.1921, Your Health; Apr.1921-Aug.1923, Health

Animal parasites and human disease,

Bibliography: p. 529-534.

Living agents of disease,

Mode of access: Internet.

The evolution of disease,

Mode of access: Internet.

Public health administration and the natural history of disease in Baltimore, Maryland, 1797-1920,

Bibliography: p. 563-565.

Global epidemiology, a geography of disease and sanitation,

"Material is based on surveys made for the Medical department of the United States army ... The surveys ... represent the work of a large number of individuals associated ... with the Medical intelligence division of the Office of the # general of the United States army."--Pref.

The Review of applied entomology.

Imprint varies: London, 1913-[1930]; Farnham Royal, Eng., [1976-]

The Practical medicine. A medical journal for the busy practitioner, specially devoted to practical diagnosis and treatment of disease by improved methods and new remedies.

Editor: 1909-10. Ram Narain.

Standard classified nomenclature of disease,

"Preliminary printing, April, 1932. Official edition, January, 1933. Second edition, January, 1935."

A digest of the law of carriers of goods and passengers by land and internal navigation.

Mode of access: Internet.

Burden of disease and injury in Aboriginal and non-Aboriginal populations in the Northern Territory

Objective: To quantify the burden of disease and injury for the Aboriginal and non-Aboriginal populations in the Northern Territory. Design and setting: Analysis of Northern Territory data for 1 January 1994 to 30 December 1998 from multiple sources. Main outcome measures: Disability-adjusted life-years (DALYs), by age, sex, cause and Aboriginality. Results: Cardiovascular disease was the leading contributor (14.9%) to the total burden of disease and injury in the NT, followed by mental disorders (14.5%) and malignant neoplasms (11.2%). There was also a substantial contribution from unintentional injury (10.4%) and intentional injury (4.9%). Overall, the NT Aboriginal population had a rate of burden of disease 2.5 times higher than the non-Aboriginal population; in the 35-54-year age group their DALY rate was 4.1 times higher. The leading causes of disease burden were cardiovascular disease for both Aboriginal men (19.1%) and women (15.7%) and mental disorders for both non-Aboriginal men (16.7%) and women (22.3%). Conclusions: A comprehensive assessment of fatal and non-fatal conditions is important in describing differentials in health status of the NT population. Our study provides comparative data to identify health priorities and facilitate a more equitable distribution of health funding.

Distribution of major health risks: findings from the global burden of disease study

Background Most analyses of risks to health focus on the total burden of their aggregate effects. The distribution of risk-factor-attributable disease burden, for example by age or exposure level, can inform the selection and targeting of specific interventions and programs, and increase cost-effectiveness. Methods and Findings For 26 selected risk factors, expert working groups conducted comprehensive reviews of data on risk-factor exposure and hazard for 14 epidemiological subregions of the world, by age and sex. Age-sex-subregion-population attributable fractions were estimated and applied to the mortality and burden of disease estimates from the World Health Organization Global Burden of Disease database. Where possible, exposure levels were assessed as continuous measures, or as multiple categories. The proportion of risk-factor-attributable burden in different population subgroups, defined by age, sex, and exposure level, was estimated. For major cardiovascular risk factors (blood pressure, cholesterol, tobacco use, fruit and vegetable intake, body mass index, and physical inactivity) 43%-61% of attributable disease burden occurred between the ages of 15 and 59 y, and 87% of alcohol-attributable burden occurred in this age group. Most of the disease burden for continuous risks occurred in those with only moderately raised levels, not among those with levels above commonly used cut-points, such as those with hypertension or obesity. Of all disease burden attributable to being underweight during childhood, 55% occurred among children 1-3 standard deviations below the reference population median, and the remainder occurred among severely malnourished children, who were three or more standard deviations below median. Conclusions Many major global risks are widely spread in a population, rather than restricted to a minority. Population-based strategies that seek to shift the whole distribution of risk factors often have the potential to produce substantial reductions in disease burden.

Assessment of disease progression in motor neuron disease

Motor neuron disease (MND) is characterised by progressive deterioration of the corticospinal tract, brainstem, and anterior horn cells of the spinal cord. There is no pathognomonic test for the diagnosis of MND, and physicians rely on clinical criteria-upper and lower motor neuron signs-for diagnosis. The presentations, clinical phenotypes, and outcomes of MND are diverse and have not been combined into a marker of disease progression. No single algorithm combines the findings of functional assessments and rating scales, such as those that assess quality of life, with biological markers of disease activity and findings from imaging and neurophysiological assessments. Here, we critically appraise developments in each of these areas and discuss the potential of such measures to be included in the future assessment of disease progression in patients with MND.