The story of Jack Ballister's fortunes : being the narrative of the adventures of a young gentleman of good family, who was kidnapped in the year 1719 and carried to the plantations of the continent of Virginia, where he fell in with that famous pirate Captain Edward Teach, or Blackbeard.

Mode of access: Internet.

Une escadre française aux Indes en 1690 : Histoire de la Compagnie royale des Indes Orientales 1664-1719 /

Mode of access: Internet.

Cartas de algunos pp. de la Compañía de Jesus sobre los sucesos de le monarquía entre los años de 1634 y 1648.

Edited by Pascual de Gayangos.

Ungedruckte, eigenhändige, vertrauliche Briefe und amtliche Schreiben aus den Jahren 1627 bis 1634 an Arnheim (v. Arnimb) Aldringer, Gallas, Piecolomini, und andere Fürsten und Feldherrn seiner Zeit. Mit einer Charakteristik des Lebens und der Feldzüge Wallensteins.

Mode of access: Internet.

The memoirs of Gregorio Panzani; : giving an account of his agency in England, in the years 1634, 1635, 1636. /

Pages [xliv] and [474] are blank.

Kitāb al-Daʻwah al-tāmmah wa-al-tadhkirah al-ʻāmmah [microform] /

Microfilm.

Brian Pendleton and his Massachusetts, 1634-1681.

Bibliographical footnotes.

The history of Windham in New Hampshire (Rockingham country). 1719-1883. A Scotch settlement (commonly called Scotch-Irish), embracing nearly one third of the ancient settlement and historic township of Londonderry, N.H.

"History of families in Windham, N.H.": p. [297]-835.

Peacham's Compleat gentleman, 1634.

With facsimile reproduction of the original title: "The compleat gentleman ... By Henry Peacham ... The second impression, much enlarged. Anno 1634"; to which is added a facsimile reproduction of the t.-p. of the 1st edition, 1622.

Historic Green Bay. 1634-1840,

Mode of access: Internet.

Histoire de mon temps. Mémoires du chancelier Pasquier

1. ptie. Révolution--consulat--empire: t. 1. 1789-1810.--t. 2. 1812-1814.--t.3. 1814-1815. 2. ptie. Restauration: t.4 1815-1820.--t. 5. 1820-1824.--t. 6. 1824-1830.

Processos naturais de biodegradação do petróleo em reservatórios

Petroleum biodegradation in reservoirs is a process caused by different microorganisms affecting many oil deposits which modifies the oil composition in a quasi-stepwise process starting from n-alkanes and isoprenoids through to diasteranes. This causes oil souring and increased viscosity, sulfur and metal content, having a direct impact on oil production and refining costs.

Phylogenetic analyses of Cladophora vagabunda (L.) C. Hoek (Cladophorales, Chlorophyta) from Brazil based on SSU rDNA sequences

The complete SSU rDNA was sequenced for 10 individuals of Cladophora vagabunda collected along the coast of Brazil. For C. rupestris (L.) Kütz. a partial SSU rDNA sequence (1634 bp) was obtained. Phylogenetic trees indicate that Cladophora is paraphyletic, but the section Glomeratae sensu lato including C. vagabunda from Brazil, Japan and France, C. albida (Nees) Kütz., C. sericea (Hudson) Kütz., and C. glomerata (L.) Kütz. is monophyletic. Within this group C. vagabunda is paraphyletic. The sequence identity for the SSU rDNA varied from 98.9% to 100% for the Brazilian C. vagabunda, and from 98.3% to 99.7% comparing the Brazilian individuals to the ones from France and Japan. Sequence identity of the Brazilian C. vagabunda to C. albida and C. sericea vary from 98.0% to 98.6%. The SSU rDNA phylogeny support partially the morphological characteristics presented by Brazilian populations of C. vagabunda. On the other hand, C. rupestris from Brazil does not group with C. rupestris from France, both sequences presenting only 96.9% of identity. The inclusion of sequences of individuals from Brazil reinforces the need of taxonomical revision for the genus Cladophora and for the complex C. vagabunda.

Lisina digestível e zinco quelado para frangos de corte machos: desempenho e retenção de nitrogênio na fase pré-inicial

Em dois ensaios experimentais, avaliaram-se níveis de lisina digestível (0,90; 1,00; 1,10; 1,20 e 1,40%) combinados a zinco quelado (43 e 253 ppm) para frangos de corte machos. Os períodos considerados foram: 1 a 11 dias de idade (desempenho) e 1 a 7 dias de idade (balanço de nitrogênio). O delineamento experimental utilizado foi de blocos ao acaso, em esquema fatorial 5 × 2. No ensaio de desempenho, utilizaram-se 7 repetições e 15 aves por unidade experimental e, no ensaio de digestibilidade, 6 repetições e 6 aves por unidade experimental. As dietas continham 2.960 de EM/kg e 21% de proteína bruta. Na fase de 1 a 11 dias de idade, não houve efeito de interação lisina digestível × zinco quelado no desempenho, mas verificou-se efeito quadrático dos níveis de lisina digestível sobre o peso final, o ganho de peso e o ganho de peso relativo e efeito linear crescente no consumo de ração. A conversão alimentar não diferiu com as variações dos níveis de lisina digestível e zinco na dieta. Nas condições de avaliação do desempenho, o nível ótimo de lisina digestível para frangos de corte machos mantidos em piso de concreto é de 1,19%. Na fase de 1 a 7 dias de idade, não houve influência da relação entre níveis de lisina digestível e zinco quelado no balanço de nitrogênio. A retenção de nitrogênio aumentou de forma linear crescente com o aumento da concentração de lisina digestível na dieta, o que está de acordo com o aumento linear do ganho de peso e da eficiência alimentar. A exigência de lisina digestível para frangos de corte machos de 1 a 7 dias de idade é igual ou superior a 1,40%.

SJL dystrophic mice express a significant amount of human muscle proteins following systemic delivery of human adipose-derived stromal cells without immunosuppression

Limb-girdle muscular dystrophies (LGMDs) are a heterogeneous group of disorders characterized by progressive degeneration of skeletal muscle caused by the absence of or defective muscular proteins. The murine model for limb-girdle muscular dystrophy 2B (LGMD2B), the SJL mice, carries a deletion in the dysferlin gene that causes a reduction in the protein levels to 15% of normal. The mice show muscle weakness that begins at 4-6 weeks and is nearly complete by 8 months of age. The possibility of restoring the defective muscle protein and improving muscular performance by cell therapy is a promising approach for the treatment of LGMDs or other forms of progressive muscular dystrophies. Here we have injected human adipose stromal cells (hASCs) into the SJL mice, without immunosuppression, aiming to assess their ability to engraft into recipient dystrophic muscle after systemic delivery; form chimeric human/mouse muscle fibers; express human muscle proteins in the dystrophic host and improve muscular performance. We show for the first time that hASCs are not rejected after systemic injection even without immunosuppression, are able to fuse with the host muscle, express a significant amount of human muscle proteins, and improve motor ability of injected animals. These results may have important applications for future therapy in patients with different forms of muscular dystrophies.