221 resultados para Benton


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Back Row: Fred Hendershot, Hepburn Ingham, Walter Nieman, Fred Rehor, James Whalen, Egmont Hildner, Cecil "Pat" Smith, Alan Boyd, James Catlett, Harold Zeiger, James Sharpe, George Washington Bixler, Otto Pobanz,

Middle Row (seated on wall): Otto Eberwein, ? Johnson, Maurice Dunne, Harry Calvin (on steps), Karl Staatz, Hoyne Howe, Clyde Bastian

Front Row: Alvin Loucks (standing), Robert Watson (on rail), John Norton, Leland Benton, captain William Cochran, Lawrence Roehm, John Maulbetsch, ? Warner (standing)

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Back Row: trainer Steve Farrell, asst. coach Germany Schulz, Thomas Bushnell (?), coach Fielding Yost

Third Row: Everett Bentley, Spencer Scott, William Cochran, Efton James, ?, Cyril Quinn, ?, Martin Galt,

Second Row: Ernest Allmendinger, Ernest Hughitt, Roy Torbet, George Patterson, Miller Pontius, James Musser, James Raynsford, ?, Frank McHale, Henry Lightner

Front Row: Jack Benton, John Lyons, Frank Millard, Robert Watson, Lawrence Roehmm Royce Traphagen

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Mode of access: Internet.

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Thesis (doctoral)--Georg-Augusts-Universitat zu Gottingen.

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"Books and journals relating to apiculture": p. 118.

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Mode of access: Internet.

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Mode of access: Internet.

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A neuronal cell line (NG115-401L-C3) was stimulated by mitogenic (angiotensin) and non-mitogenic (bradykinin) peptides and examined for the time course of changes in the levels of radiolabelled inositol phosphates and phospholipids. Both peptides stimulated the time-dependent production of Ins(1,4,5)P3 and related metabolites. Bradykinin caused a much larger increase in Ins(1,4,5)P3 than did angiotensin. However, both peptides stimulated similar rises in the levels of Ins(1,3,4)P3 and InsP4. Bradykinin but not angiotensin, caused a rapid (within 2 s) fall in the levels of PtdIns(4,5)P2 and PtdIns(4)P. Serum pretreatment of the cells caused a 2-3-fold potentiation of both the responses to bradykinin and angiotensin. Although significant levels of PtdIns(3)P were detected in resting cells neither mitogenic (angiotensin, insulin-like growth factor I, transforming growth factor beta) nor non-mitogenic (bradykinin, nerve growth factor interleukin-1) receptor activation changed its levels, arguing against regulation of either PtdIns 3-kinase or PtdIns(3)P phosphatase. We conclude that, as judged by the levels of its product. PtdIns(3)P, the enzyme PtdIns 3-kinase is not activated. This questions the significance of this activity in the receptor-mediated initiation of DNA synthesis.

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The application of any e-Solution promises significant returns. In particular, using internet technologies both within enterprises and across the supply (value) chain provides real opportunity, not only for operational improvement but also for innovative strategic positioning. However, significant questions obscure potential investment; how any value will actually be created and, importantly, how this value will be shared across the value chain is not clear. This paper will describe a programme of research that is developing an enterprise simulator that will provide a more fundamental understanding of the impact of e-Solutions across operational supply chains, in terms of both standard operational and financial measures of performance. An efficient supply chain reduces total costs of operations by sharing accurate real-time information and coordinating inter-organizational business processes. This form of electronic link between organizations is known as business-to-business (B2B) e-Business. The financial measures go beyond simple cost calculations to real bottom-line performance by modelling the financial transactions that business processes generate. The paper will show how this enterprise simulator allows for a complete supply chain to be modelled in this way across four key applications: control system design, virtual enterprises, pan-supply-chain performance metrics and supporting e-Supply-chain design methodology.