Enumeratio plantarum Horti regii berolinensis. post mortem autores editum.

Pref. in German, signed v. Schlechtendal.

Johannis Schneidevini ... In quatuor Institutionum imperialium D. Justiniani libros, commentarii nunc post mortem ejus ... cum multis libellorum, & actuum judicialium formis, atq́ue juris saxonici consensu & antinomia, editi.

In double columns.

Flora ægyptiaco-arabica. Sive Descriptiones plantarum, quas per Ægyptum inferiorem et Arabiam felicem detexit, illustravit Petrus Forskål ... Post mortem auctoris edidit Carsten Niebuhr. Accedit tabula Arabiae felicis geographico-botanica.

Florula littoris Galliæ ad Estac prope Massiliam. Accedit florula insulæ Maltæ. p., [i]-xiv.--Flora constantinopolitana, littoris ad Dardenellos et insularum Tenedos, Imros, Rhodi, p. [xv]-xxxvi.--Flora ægyptiaca: sive Catalogus plantarum systematicus Ægypti inferioris: Alexandriæ, Rosettæ, Kahiræ, Sues, p. [xxxvii]-lxxviii.--Flora arabicovemen. Sive Catalogue plantarum Arabiæ felicis systematicus. p. [lxxix]-cxxvi.--Descriptiones plantarum floræ ægyptiaco-arabicæ. p. 1-219.

A catalogue of the inquisitions post mortem for the county of York, for the reigns of James I. and Charles I., in the Courts of Chancery and of Wards and Liveries, the latter presented by John Sykes.

Bound with: A catalogue of the Yorkshire wills at Somerset House ....

Abstracts of Gloucestershire Inquisitiones post mortem returned into the Court of Chancery.

Parts 4-6 have title: Abstracts in Inquisitiones post mortem for Gloucestershire.

Post-mortem use of wealth, including a consideration of ante-mortem gifts;

Mode of access: Internet.

A calendar of post mortem inquisitions relating to the county of Sussex, 1 to 25 Elizabeth.

Mode of access: Internet.

Post-mortem examinations of wild birds and mammals /

"May 1937."

Abstracts of Inquisitiones post mortem relating to the city of London, returned into the Court of chancery during the Tudor period.

Parts 4- have title: Abstracts of Inquisitiones post mortem for Gloucestershire.

A text-book of pathology with an introductory section on post-mortem examinations and the methods of preserving and examining diseased tissues.

Bibliography at ends of some chapters.

A microarray study of post-mortem mRNA degradation in mouse brain tissue

Background: Although there is evidence that post-mortem interval (PMI) is not a major contributor to reduced overall RNA integrity, it may differentially affect a subgroup of gene transcripts that are susceptible to PMI-related degradation. This would particularly have ramifications for microarray studies that include a broad spectrum of genes. Method: Brain tissue was removed from adult mice at 0, 6, 12, 18, 24,36 and 48 h post-mortem. RNA transcript abundance was measured by hybridising RNA from the zero time point with test RNA from each PMI time point, and differential gene expression was assessed using cDNA microarrays. Sequence and ontological analyses were performed on the group of RNA transcripts showing greater than two-fold reduction. Results: Increasing PMI was associated with decreased tissue pH and increased RNA degradation as indexed by 28S/18S ribosomal RNA ratio. Approximately 12% of mRNAs detected on the arrays displayed more than a two-fold decrease in abundance by 48 It post-mortem. An analysis of nucleotide composition provided evidence that transcripts with the AUUUA motif in the 3' untranslated region (3'UTR) were more susceptible to PMI-related RNA degradation, compared to transcripts not carrying the 3'UTR AUUUA motif. Consistent with this finding, ontological analysis showed transcription factors and elements to be over-represented in the group of transcripts susceptible to degradation. Conclusion: A subgroup of mammalian mRNA transcripts are particularly susceptible to PMI-related degradation, and as a group, they are more likely to carry the YUTR AUUUA motif. PMI should be controlled for in human and animal model post-mortem brain studies, particularly those including a broad spectrum of mRNA transcripts. (c) 2005 Elsevier B.V. All rights reserved.

Responses in carcass lean pH at 24 hours post-mortem in lines of Large White pigs selected for growth rate on a fixed ration over a set time

A restricted maximum likelihood analysis applied to an animal model showed no significant differences (P > 0.05) in pH value of the longissimus dorsi measured at 24 h post-mortem (pH24) between high and low lines of Large White pigs selected over 4 years for post-weaning growth rate on restricted feeding. Genetic and phenotypic correlations between pH24 and production and carcass traits were estimated using all performance testing records combined with the pH24 measurements (5.05–7.02) on slaughtered animals. The estimate of heritability for pH24 was moderate (0.29 ± 0.18). Genetic correlations between pH24 and production or carcass composition traits, except for ultrasonic backfat (UBF), were not significantly different from zero. UBF had a moderate, positive genetic correlation with pH24 (0.24 ± 0.33). These estimates of genetic correlations affirmed that selection for increased growth rate on restricted feeding is likely to result in limited changes in pH24 and pork quality since the selection does not put a high emphasis on reduced fatness.

Genetic polymorphisms in post-mortem alcoholics

We investigated the hypothesis that alcoholism risk may be mediated by genes for neurotransmitters (dopamine, serotonin, opioid, GABAA and glutamate) associated with the dopamine reward system, and with genes involved in ethanol metabolism and fibrogenesis (ADH2, ADH3, ALDH2, CYP2E1, COL1A2, and ApoE). DNA was extracted from brain tissue collected at autopsy from pathologically characterised alcoholics and controls. PCR-based studies showed that alcoholism was associated with polymorphisms of the dopamine D2 receptor (DRD2) Taq1 B (p 0.005) and the GABAA 2 subunit C1412T (p 0.007) genes but not with the glutamate receptor subunit gene NR2B (366C/G), the serotonin transporter gene (5HTTL-PR), the dopamine transporter gene DAT1(SLC6A3), the Mu opioid receptor gene MOR1 (A118G and C1031G), the dopamine D2 receptor gene DRD2 Taq1 A or the GABAA 1(A15G), 6(T1519C) and 2(G3145A) subunit genes. The glial glutamate transporter gene EAAT2 polymorphism G603A was associated with alcoholic cirrhosis (p 0.024). The genotype for the most active alcohol dehydrogenase ADH3 was associated with a lower risk of alcoholism (p 0.027) and was less prevalent in alcoholics with DRD2 Taq1 A2/A2 (p 0.007), Taq1 B2/B2 (p 0.038) and GABAA-2 1412C/C (p 0.005) and EAAT2 603G/A (p 0.020) genotypes. Combined genotypes of DRD2 Taq1 A and B, GABAA-2, and EAAT2 G603A polymorphisms suggested a concerted influence of dopamine, GABAA and glutamatergic neurotransmitters in the predisposition to alcoholism.