Comercio espiritual en fe de la Iglesia militante con la triunfante: practicado, y explicado en la vida, y virtudes de ... Sor Josepha Berride, de la tercera orden de Santo Domingo /

Mode of access: Internet.

Slownjk česko-némecký /

Accompanied by supplement "Dodavky ke Slovníku Josefa Jungmanna" (66 p. ; 29 cm.9), published in 1851.

Ejemplar de religiosas : Vida de la M. R. M. Sor María Josefa Lina de la Sma. Trinidad, fundadora del Convento de la Purísima Concepción en la Ciudad de San Miguel...

UANL

Ejemplar de religiosas : vida de la muy reverenda Madre Sor María Josefa Lino de la Santísima Trinidad...

UANL

La Célebre década de Bilbao : ó sea, memoria de los festejos con que su muy ilustre Ayuntamiento ha procurado obsequiar a SS. MM. los Reyes nuestros Señores, D. Fernando Séptimo y Dña. Josefa María Amalia.

Marca tip. en port.

La Célebre década de Bilbao : ó sea, memoria de los festejos con que su muy ilustre Ayuntamiento ha procurado obsequiar a SS. MM. los Reyes nuestros Señores, D. Fernando Séptimo y Dña. Josefa María Amalia.

Marca tip. en port.

Encomios funebres ascetico-panegiricos, que en las honras, i exequias de ... Josefa Benlloch

Sign. : [calderón]4, 2[calderón]2, A-I4, K2

Rada wsselikých zwiřat nerozumných neb zhowadilých, y ptactwa, kterau člowěku wsselikého powoláni dáwagi, w ćemby gegich přirozeni následowati, a gakých powah k zpráwě žiwota dobrého se wystřihati měl. Od Giřiho Melantrycha z Awentýnu. Kniha vžitečná y kratochwilná.

"Předmluva" signed J.D. [i.e. Josef Dobrovský]

Slovenská přísloví, pořekadla a úsloví /

Mode of access: Internet.

Přśpěvky k životu a dílu Jos. Dobrovského.

"Řada 2. napsali Josef Volf a Miloš B. Volf."

Starožitnosti země české. díl 1[-3, sv. 1. Na základě praehistorické stírky Musea král. českého, píse J. L. Píč.

Bibliographical footnotes.

Evaluación de la estrategia en manejo de conflictos de la PNGIRH. Estudio de caso: territorio colectivo de doña Josefa, municipio de Atrato, Chocó

El objeto de este trabajo es evaluar la estrategia en manejo de conflictos de la política nacional de gestión integral del recurso hídrico en el corregimiento de doña Josefa, departamento del Chocó en función de sus actores.

Pyrimidine-5'-nucleotidase Campinas, A New Mutation (p.r56g) In The Nt5c3 Gene Associated With Pyrimidine-5'-nucleotidase Type I Deficiency And Influence Of Gilbert's Syndrome On Clinical Expression.

Pyrimidine-5'-nucleotidase type I (P5'NI) deficiency is an autosomal recessive condition that causes nonspherocytic hemolytic anemia, characterized by marked basophilic stippling and pyrimidine nucleotide accumulation in erythrocytes. We herein present two African descendant patients, father and daughter, with P5'N deficiency, both born from first cousins. Investigation of the promoter polymorphism of the uridine diphospho glucuronosyl transferase 1A (UGT1A) gene revealed that the father was homozygous for the allele (TA7) and the daughter heterozygous (TA6/TA7). P5'NI gene (NT5C3) gene sequencing revealed a further change in homozygosity at amino acid position 56 (p.R56G), located in a highly conserved region. Both patients developed gallstones; however the father, who had undergone surgery for the removal of stones, had extremely severe intrahepatic cholestasis and, liver biopsy revealed fibrosis and siderosis grade III, leading us to believe that the homozygosity of the UGT1A polymorphism was responsible for the more severe clinical features in the father. Moreover, our results show how the clinical expression of hemolytic anemia is influenced by epistatic factors and we describe a new mutation in the P5'N gene associated with enzyme deficiency, iron overload, and severe gallstone formation. To our knowledge, this is the first description of P5'N deficiency in South Americans.

Preserved Fertility In A Patient With Gynecomastia Associated With The P.pro695ser Mutation In The Androgen Receptor.

The androgen insensitivity syndrome (AIS) is described as a dysfunction of the androgen receptor (AR) in 46,XY individuals, which can be associated with mutations in the AR gene or can be due to unknown mechanisms. Different mutations in AIS generally cause variable phenotypes that range from a complete hormone resistance to a mild form usually associated with male infertility. The purpose of this study was to search for mutations in the AR gene in a fertile man with gynecomastia and to evaluate the influence of the mutation on the AR transactivation ability. Sequencing of the AR gene revealed the p.Pro695Ser mutation. It is located within the AR ligand-binding domain. Bioinformatics analysis indicated a deleterious role, which was verified after testing transactivation activity and N-/C-terminal (N/C) interaction by in vitro expression of a reporter gene and 2-hybrid assays. p.Pro695Ser showed low levels of both transactivation activity and N/C interaction at low dihydrotestosterone (DHT) conditions. As the ligand concentration increased, both transactivation activity and N/C interaction also increased and reached normal levels. Therefore, this study provides functional insights for the p.Pro695Ser mutation described here for the first time in a patient with mild AIS. The expression profile of p.Pro695Ser not only correlates to the patient's phenotype, but also suggests that a high-dose DHT therapy may overcome the functional deficit of the mutant AR.