260 resultados para FEMORIS MUSCLE TEAR

Elevated glucose concentration potentiates vascular smooth muscle cell chernotaxis via phosphoinositide 3-kinase-p110 beta, protein kinase B and protein kinase C beta II

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High glucose mediates vascular smooth muscle cell survival through the activation of protein kinase B and extracellular regulated kinase 1/2.

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Vascular smooth muscle cell chemotaxis is potentiated by glucose via phosphoinositide 3-kinase-p110 beta, Akt, ERK1/2 and protein kinase C beta 11 dependent pathways.

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Effects of glucose and alpha-lipoic acid on glutathione levels, DNA damage and apoptotic proteins in smooth muscle cells.

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Glucose-potentiated vascular smooth muscle cell chemotaxis is mediated by phospholnositide-3-kinase-p110 beta

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Platelet derived growth Factor-B induced chemotaxis in vascular smooth muscle cells: Glucose potentiation

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Glucose-potentiated vascular smooth muscle cell chemotaxis to fetal calf serum is mediated by PI3Kinase and MAPKinase

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Chemotaxis and oxidative stress in vascular smooth muscle: Effects of long chain fatty acids and glucose.

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Vascular smooth muscle cell and macrophage chemotaxis: Modulation by glucose

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Effects of glucose and fatty acids on antioxidant defences and function in porcine vascular smooth muscle cells (VSMC) in vitro

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Variation in Epithelium and Muscle in Two Strains of Mice.

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Beta-amyloid protein protein precursor expression in lacrimal glands and tear fluid.

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[Regenerative calcium-induced liberation of calcium from intracellular stores in smooth muscle cells]

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[Inhibitory effect of caffeine on the potential-dependent calcium current in isolated intestinal smooth muscle cells]

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Potassium currents in the isolated single smooth muscle cell membrane

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