Biblioteca Digital

81 resultados para arritmia ventricular

Use of the cyclic AMP antagonist, Rp-cAMPS, to provide evidence that secretin exerts a contractile action on ventricular cardiomyocytes by a cyclic AMP-dependent mechanism

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Involvement of L-type calcium channels in contractile responses to secretin and VIP but not CGRP in ventricular cardiomyocytes

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Amylin stimulates a positive contractile response in ventricular cardiomyocytes via CGRP preferring receptors

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Hypertrophic effects of CGRP and amylin on adult ventricular cardiomyocytes

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Involvement of ETA and ETB receptors in the hypertrophic effects of endothelin-1 in adult ventricular cardiomyocytes

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Multiple signalling pathways associated with receptors for somatostatin-14 on ventricular cardiomyocytes

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Use of myoinositol 1,26 trisphosphate pp56 to distinguish between phospholipase C-dependent and phospholipase C-independent contractile coupling mechanisms in mammalian ventricular cardiomyocytes

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Contribution of de novo protein synthesis to the hypertrophic effects of IGF-1 and thyroid hormone in adult ventricular cardiomyocytes

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Involvement of Y1 and Y2 receptors in the hypertrophic response to neuropeptide Y in mammalian ventricular cardiomyocytes

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The thiazolidinedione, troglitazone, does not initiate ventricular cardiomyocyte hypertrophy in vitro but may sensitise cardiomyocytes to the hypertrophic effects of serum growth factors

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Altered functional responsiveness to neuropeptide Y acting at Y2 receptors in established ventricular cell hypertrophy in spontaneously hypertensive rat

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Evidence for enhanced endothelin ETB receptor affinity during the progression of hypertension-induced ventricular cardiomyocyte hypertrophy

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Altered ETB receptor-mediated signalling in ventricular cardiomyocytes in response to pressure overload

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Possible trophic effects of a metabolite of BRL49653C, SB271258, on adult ventricular cardiomyocytes in vitro.

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ETA receptor-mediated contractile function is depressed in left ventricular cardiomyocyte hypertrophy and the response does not alter following transition to decompensated heart failure

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