Migration: A Heideggerian analysis

The phenomenon of migration has been widely researched by the social sciences. Theories regarding the migrant have been developed in terms of the oppressive social context that is often encountered, proposing different alternatives to understand and overcome such oppression. Through the current project, an alternative view is presented that first, questions the accuracy of the social theories of migration and second, proposes an alternative understanding of this experience. Martin Heidegger’s phenomenology of Being offers a contextualized view of existence that nonetheless includes elements of our experience that are shared due to a common mode of being. I use Heidegger’s philosophy in order to broaden the understanding of the migrant’s experience analyzing those elements that he identifies as shared (for instance: human sociability, a desire for a home, the uncanny, etc.) and comparing them with common issues raised by migrants (identity, homesickness, belonging, etc.). In this way, I intend to present a more complete picture of the experience of migration that considers both empirical evidence of individual migrants and an existential analysis that incorporates the defining elements of our world and our existence as crucial means to understand any experience, including that of migration.

Growing up in a bubble: using germ-free animals to assess the influence of the gut microbiota on brain and behavior

There is a growing recognition of the importance of the commensal intestinal microbiota in the development and later function of the central nervous system. Research using germ-free mice (mice raised without any exposure to microorganisms) has provided some of the most persuasive evidence for a role of these bacteria in gut-brain signalling. Key findings show that the microbiota is necessary for normal stress responsivity, anxiety-like behaviors, sociability, and cognition. Furthermore, the microbiota maintains central nervous system homeostasis by regulating immune function and blood brain barrier integrity. Studies have also found that the gut microbiota influences neurotransmitter, synaptic, and neurotrophic signalling systems and neurogenesis. The principle advantage of the germ-free mouse model is in proof-of-principle studies and that a complete microbiota or defined consortiums of bacteria can be introduced at various developmental time points. However, a germ-free upbringing can induce permanent neurodevelopmental deficits that may deem the model unsuitable for specific scientific queries that do not involve early-life microbial deficiency. As such, alternatives and complementary strategies to the germ-free model are warranted and include antibiotic treatment to create microbiota-deficient animals at distinct time points across the lifespan. Increasing our understanding of the impact of the gut microbiota on brain and behavior has the potential to inform novel management strategies for stress-related gastrointestinal and neuropsychiatric disorders.