2 resultados para EXTENDED AMYGDALA

em Boston University Digital Common


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We investigate numerically the ground state phase diagram of the one-dimensional extended Hubbard model, including an on--site interaction U and a nearest--neighbor interaction V. We focus on the ground state phases of the model in the V >> U region, where previous studies have suggested the possibility of dominant superconducting pairing fluctuations before the system phase separates at a critical value V=V_PS. Using quantum Monte Carlo methods on lattices much larger than in previous Lanczos diagonalization studies, we determine the boundary of phase separation, the Luttinger Liquid correlation exponent K_rho, and other correlation functions in this region. We find that phase separation occurs for V significantly smaller than previously reported. In addition, for negative U, we find that a uniform state re-enters from phase separation as the electron density is increased towards half filling. For V < V_PS, our results show that superconducting fluctuations are not dominant. The system behaves asymptotically as a Luttinger Liquid with K_rho < 1, but we also find strong low-energy (but gapped) charge-density fluctuations at a momentum not expected for a standard Luttinger Liquid.

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A neural model is presented that explains how outcome-specific learning modulates affect, decision-making and Pavlovian conditioned approach responses. The model addresses how brain regions responsible for affective learning and habit learning interact, and answers a central question: What are the relative contributions of the amygdala and orbitofrontal cortex to emotion and behavior? In the model, the amygdala calculates outcome value while the orbitofrontal cortex influences attention and conditioned responding by assigning value information to stimuli. Model simulations replicate autonomic, electrophysiological, and behavioral data associated with three tasks commonly used to assay these phenomena: Food consumption, Pavlovian conditioning, and visual discrimination. Interactions of the basal ganglia and amygdala with sensory and orbitofrontal cortices enable the model to replicate the complex pattern of spared and impaired behavioral and emotional capacities seen following lesions of the amygdala and orbitofrontal cortex.