Monoaminergic Neuropathology in Alzheimer's disease
| Contribuinte(s) |
University of Aberdeen, Medicine, Medical Sciences & Nutrition, Medical Sciences University of Aberdeen, Medicine, Medical Sciences & Nutrition, Applied Medicine |
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| Data(s) |
05/08/2016
05/08/2016
12/04/2016
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| Resumo |
Acknowledgments This work was supported by The Croatian Science Foundation grant. no. IP-2014-09-9730 (“Tau protein hyperphosphorylation, aggregation, and trans-synaptic transfer in Alzheimer’s disease: cerebrospinal fluid analysis and assessment of protective neuroprotective compounds”) and European Cooperation in Science and Technology (COST) Action CM1103 (“Stucture-based drug design for diagnosis and treatment of neurological diseases: dissecting and modulating complex function in the monoaminergic systems of the brain”). PRH is supported in part by NIH grant P50 AG005138. Peer reviewed Postprint |
| Identificador |
Šimić , G , Leko , M B , Wray , S , Harrington , C R , Delalle , I , Jovanov-Milošević , N , Bažadona , D , Buée , L , Silva , R D , Giovanni , G D , Wischik , C M & Hof , P R 2016 , ' Monoaminergic Neuropathology in Alzheimer's disease ' Progress in Neurobiology . , 10.1016/j.pneurobio.2016.04.001 0301-0082 PURE: 66818067 PURE UUID: 8177319c-1ac3-49d6-a970-3681589d2b6a PubMed: 27084356 |
| Idioma(s) |
eng |
| Relação |
Progress in Neurobiology |
| Direitos |
© 2016. This manuscript version is made available under the CC-BY-NC-ND 4.0 license http://creativecommons.org/licenses/by-nc-nd/4.0/ |
| Palavras-Chave | #5-hydroxytryptamine (serotonin) #Alzheimer's disease #amyloid beta (Aβ) protein #blood-brain barrier #cerebrospinal fluid #epigenetics #locus coeruleus #metals #monoamines #neurofibrillary degeneration #non-cognitive symptoms #nucleus raphe dorsalis #phosphorylation #sleep-wake cycle #tau protein #RC0321 Neuroscience. Biological psychiatry. Neuropsychiatry #RC0321 |
| Tipo |
Journal article |
