A new light on an old disease: adhesion signaling in pemphigus vulgaris.


Autoria(s): Galichet, Arnaud; Borradori, Luca; Müller, Eliane Jasmine
Data(s)

01/01/2014

Resumo

Disruption of desmosomal cadherin adhesion leads to the activation of intracellular signaling pathways that are responsible for blister formation in pemphigus vulgaris (PV). Recent studies corroborate the implication of the p38 mitogen-activated protein kinase in PV blistering via its downstream effector mitogen-activated protein kinase activated protein kinase 2. These insights highlight the key role of cadherins in tissue homeostasis and are expected to change pemphigus management.

Formato

application/pdf

Identificador

http://boris.unibe.ch/59186/1/2014_borradori_j%20invest%20dermatol_2.pdf

Galichet, Arnaud; Borradori, Luca; Müller, Eliane Jasmine (2014). A new light on an old disease: adhesion signaling in pemphigus vulgaris. Journal of Investigative Dermatology, 134(1), pp. 8-10. Nature Publishing 10.1038/jid.2013.439 <http://dx.doi.org/10.1038/jid.2013.439>

doi:10.7892/boris.59186

info:doi:10.1038/jid.2013.439

info:pmid:24352077

urn:issn:0022-202X

Idioma(s)

eng

Publicador

Nature Publishing

Relação

http://boris.unibe.ch/59186/

Direitos

info:eu-repo/semantics/restrictedAccess

Fonte

Galichet, Arnaud; Borradori, Luca; Müller, Eliane Jasmine (2014). A new light on an old disease: adhesion signaling in pemphigus vulgaris. Journal of Investigative Dermatology, 134(1), pp. 8-10. Nature Publishing 10.1038/jid.2013.439 <http://dx.doi.org/10.1038/jid.2013.439>

Palavras-Chave #630 Agriculture #610 Medicine & health
Tipo

info:eu-repo/semantics/article

info:eu-repo/semantics/publishedVersion

PeerReviewed