A new light on an old disease: adhesion signaling in pemphigus vulgaris.
Data(s) |
01/01/2014
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Resumo |
Disruption of desmosomal cadherin adhesion leads to the activation of intracellular signaling pathways that are responsible for blister formation in pemphigus vulgaris (PV). Recent studies corroborate the implication of the p38 mitogen-activated protein kinase in PV blistering via its downstream effector mitogen-activated protein kinase activated protein kinase 2. These insights highlight the key role of cadherins in tissue homeostasis and are expected to change pemphigus management. |
Formato |
application/pdf |
Identificador |
http://boris.unibe.ch/59186/1/2014_borradori_j%20invest%20dermatol_2.pdf Galichet, Arnaud; Borradori, Luca; Müller, Eliane Jasmine (2014). A new light on an old disease: adhesion signaling in pemphigus vulgaris. Journal of Investigative Dermatology, 134(1), pp. 8-10. Nature Publishing 10.1038/jid.2013.439 <http://dx.doi.org/10.1038/jid.2013.439> doi:10.7892/boris.59186 info:doi:10.1038/jid.2013.439 info:pmid:24352077 urn:issn:0022-202X |
Idioma(s) |
eng |
Publicador |
Nature Publishing |
Relação |
http://boris.unibe.ch/59186/ |
Direitos |
info:eu-repo/semantics/restrictedAccess |
Fonte |
Galichet, Arnaud; Borradori, Luca; Müller, Eliane Jasmine (2014). A new light on an old disease: adhesion signaling in pemphigus vulgaris. Journal of Investigative Dermatology, 134(1), pp. 8-10. Nature Publishing 10.1038/jid.2013.439 <http://dx.doi.org/10.1038/jid.2013.439> |
Palavras-Chave | #630 Agriculture #610 Medicine & health |
Tipo |
info:eu-repo/semantics/article info:eu-repo/semantics/publishedVersion PeerReviewed |