Protein kinase C{delta} deficiency accelerates neointimal lesions of mouse injured artery involving delayed reendothelialization and vasohibin-1 accumulation
Data(s) |
2010
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Resumo |
To use protein kinase C (PKC) d-knockout mice to investigate the role of PKCd in lesion development and to understand the underlying mechanism of the vascular disease. |
Identificador | |
Idioma(s) |
eng |
Direitos |
info:eu-repo/semantics/restrictedAccess |
Fonte |
Bai , X , Margariti , A , Hu , Y , Sato , Y , Zeng , L , Ivetic , A , Habi , O , Mason , J C , Wang , X & Xu , Q 2010 , ' Protein kinase C{delta} deficiency accelerates neointimal lesions of mouse injured artery involving delayed reendothelialization and vasohibin-1 accumulation ' Arteriosclerosis Thrombosis and Vascular Biology , vol 30 , no. 12 , pp. 2467-74 . DOI: 10.1161/ATVBAHA.110.215723 |
Tipo |
article |