Protein kinase C{delta} deficiency accelerates neointimal lesions of mouse injured artery involving delayed reendothelialization and vasohibin-1 accumulation


Autoria(s): Bai, Xue; Margariti, Andriana; Hu, Yanhua; Sato, Yasufumi; Zeng, Lingfang; Ivetic, Aleksandar; Habi, Ouassila; Mason, Justin C; Wang, Xian; Xu, Qingbo
Data(s)

2010

Resumo

To use protein kinase C (PKC) d-knockout mice to investigate the role of PKCd in lesion development and to understand the underlying mechanism of the vascular disease.

Identificador

http://pure.qub.ac.uk/portal/en/publications/protein-kinase-cdelta-deficiency-accelerates-neointimal-lesions-of-mouse-injured-artery-involving-delayed-reendothelialization-and-vasohibin1-accumulation(efb56406-3194-4c53-b450-af4dd027a0fe).html

http://dx.doi.org/10.1161/ATVBAHA.110.215723

Idioma(s)

eng

Direitos

info:eu-repo/semantics/restrictedAccess

Fonte

Bai , X , Margariti , A , Hu , Y , Sato , Y , Zeng , L , Ivetic , A , Habi , O , Mason , J C , Wang , X & Xu , Q 2010 , ' Protein kinase C{delta} deficiency accelerates neointimal lesions of mouse injured artery involving delayed reendothelialization and vasohibin-1 accumulation ' Arteriosclerosis Thrombosis and Vascular Biology , vol 30 , no. 12 , pp. 2467-74 . DOI: 10.1161/ATVBAHA.110.215723

Tipo

article