Mechanism of phosphatidylinositol 3-kinase-dependent increases in BAC1.2F5 macrophage-like cell density in response to M-CSF: phosphatidylinositol 3-kinase inhibitors increase the rate of apoptosis rather than inhibit DNA synthesis.


Autoria(s): Murray, James
Data(s)

2000

Identificador

http://pure.qub.ac.uk/portal/en/publications/mechanism-of-phosphatidylinositol-3kinasedependent-increases-in-bac12f5-macrophagelike-cell-density-in-response-to-mcsf-phosphatidylinositol-3kinase-inhibitors-increase-the-rate-of-apoptosis-rather-than-inhibit-dna-synthesis(0a0aa6cc-01c2-4517-a3b6-7543d423e0c1).html

http://www.scopus.com/inward/record.url?scp=0033679303&partnerID=8YFLogxK

Idioma(s)

eng

Direitos

info:eu-repo/semantics/restrictedAccess

Fonte

Murray , J 2000 , ' Mechanism of phosphatidylinositol 3-kinase-dependent increases in BAC1.2F5 macrophage-like cell density in response to M-CSF: phosphatidylinositol 3-kinase inhibitors increase the rate of apoptosis rather than inhibit DNA synthesis. ' Inflammation research : official journal of the European Histamine Research Society ... [et al.] , vol 49(11) , no. 11 , pp. 610-618 .

Palavras-Chave #/dk/atira/pure/subjectarea/asjc/3000/3005 #Toxicology #/dk/atira/pure/subjectarea/asjc/2700/2736 #Pharmacology (medical) #/dk/atira/pure/subjectarea/asjc/2400/2403 #Immunology #/dk/atira/pure/subjectarea/asjc/1300/1307 #Cell Biology
Tipo

article