77 resultados para exercise performance

em Repositório Institucional UNESP - Universidade Estadual Paulista "Julio de Mesquita Filho"


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The aim of this study was to investigate the effects of preferred and nonpreferred music on exercise distance, Heart Rate (HR), and Rating of Perceived Exertion (RPE) during continuous cycling exercise performed at high intensity Fifteen participants performed five test sessions During two sessions, they cycled with fixed workload on ergometer to determine the Critical Power (Cl') intensity Then, they performed three more sessions cycling at CP intensity listening to Preferred Music, listening to Nonpreferred Music, and No Music The HR responses in the exercise sessions did not differ among all conditions However, the RPE was higher for Nonpreferred Music than in the other conditions The performance under Preferred Music (9 8 +/- 4 6km) was greater than under Nonpreferred Music (7 1 +/- 3 5km) conditions Therefore, listening to Preferred Music during continuous cycling exercise at high intensity can Increase the exercise distance, and individuals listening to Nonpreferred Music can perceive more discomfort caused by the exercise

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Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)

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Dyer and McKune (2013) stated that music tempo has no influence on performance, physiological, and psychophysical variables in well-trained cyclists during high intensity endurance tasks. However, there are important limitations in the methodology of the study. The participants'music preferences and tempo change were not well measured. It is not possible to affirm that music tempo does not influence athletes'performance. Potential areas of future research include: (a) use of instruments to assess the qualities of music; (b) standardizing music of tempo according to exercise type (e.g., running, cycling, etc.); (c) considering training level of the participants (i.e., athletes and non-athletes); and (d) use of instruments to assess concentration during exercise.

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The present study examined the interaction of hypercaloric diet (HD) and physical exercise on lipid profile and oxidative stress in serum and liver of rats. Male Wistar rats (60-days-old) were fed with a control (C) and hypercaloric diet (H). Each of the two dietary groups (C and H) was divided into three subgroups (n = 8), sedentary (CS and HS), exercised 2 days a week (CE2 and HE2) and exercised 5 days a week (CE5 and HE5). The swimming was selected as a model for exercise performance. After 8-weeks exercised rats showed decreased lactate dehydrogenase serum activities, demonstrating the effectiveness of the swimming as an aerobic-training protocol. Exercise 5-days a week reduced the body weight gain. Triacylglycerol (TG) and very low-density lipoprotein (VLDL-C) were increased in HD-fed rats. HE5 and CE5 rats had decreased TG, VLDL-C and cholesterol. HE2 rats had enhanced high-density lipoprotein (HDL-C) in serum. No alterations were observed in lipid hydroperoxide (LH), while total antioxidant substances (TAS) were increased in serum of exercised rats. HD-fed rats had hepatic TG accumulation. Superoxide dismutase activities were increased and catalase was decreased in liver of exercised rats. The interaction of HD and physical exercise reduced TAS and enhanced LH levels in hepatic tissue. In conclusion, this study confirmed the beneficial effect of physical exercise as a dyslipidemic-lowering component. Interaction of HD and physical exercise had discrepant effects on serum and liver oxidative stress. The interaction of HID and physical exercise reduced the oxidative stress in serum. HD and physical exercise interaction had pro-oxidant effect on hepatic tissue, suggesting that more studies should be done before using physical exercise as an adjunct therapy to reduce the adverse effects of HD. (c) 2006 Elsevier Ltd. All rights reserved.

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Controversy still exists concerning the potential ergogenic benefit of caffeine (CAF) for exercise performance. The purpose of this study was to compare the effects of CAF ingestion on endurance performance during exercise on a bicycle ergometer at two different intensities, i.e., approximately 10% below and 10% above the anaerobic threshold (AT). Eight untrained males, non-regular consumers of CAF, participated in this study. AT, defined as the intensity (watts) corresponding to a lactate concentration of 4 mM, was determined during an incremental exercise test from rest to exhaustion on an electrically braked cycle ergometer. on the basis of these measurements, the subjects were asked to cycle until exhaustion at two different intensities, i.e., approximately 10% below and 10% above AT. Each intensity was performed twice in a double-blind randomized order by ingesting either CAF (5 mg/kg) or a placebo (PLA) 60 min prior to the test. Venous blood was analyzed for free fatty acid, glucose, and lactate, before, during, and immediately after exercise. Rating of perceived exertion and time to exhaustion were also measured during each trial. There were no differences in free fatty acids or lactate levels between CAF and PLA during and immediately after exercise for either intensity. Immediately after exercise glucose increased in the CAF trial at both intensities. Rating of perceived exertion was significantly lower (CAF = 14.1 ± 2.5 vs PLA = 16.6 ± 2.4) and time to exhaustion was significantly higher (CAF = 46.54 ± 8.05 min vs PLA = 32.42 ± 14.81 min) during exercise below AT with CAF. However, there was no effect of CAF treatment on rating of perceived exertion (CAF = 18.0 ± 2.7 vs PLA = 17.6 ± 2.3) and time to exhaustion (CAF = 18.45 ± 7.28 min vs PLA = 19.17 ± 4.37 min) during exercise above AT. We conclude that in untrained subjects caffeine can improve endurance performance during prolonged exercise performed below AT and that the decrease of perceived exertion can be involved in this process

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The mechanisms underpinning fatigue and exhaustion, and the specific sources of exercise-endurance intensity regulation and (in)tolerance have been investigated for over a century. Although several scientific theories are currently available, over the past five years a new framework called Psychobiological model has been proposed. This model gives greater attention to perceptual and motivational factors than its antecedents, and their respective influence on the conscious process of decision-making and behavioral regulation. In this review we present experimental evidences and summarize the key points of the Psychobiological model to explain intensity regulation and (in)tolerance in endurance exercise. Still, we discuss how the Psychobiological model explains training-induced adaptations related to improvements in performance, experimental manipulations, its predictions, and propose future directions for this investigative area. The Psychobiological model may give a new perspective to the results already published in the literature, helping scientists to better guide their research problems, as well as to analyze and interpret new findings more accurately.

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Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)

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Altos desempenhos esportivos demandam treinamentos pesados necessários ao estímulo adaptativo específico a cada esporte. A elevada carga de treino é geralmente acompanhada de discreta fadiga e reduções agudas no desempenho, mas caso acompanhada de períodos apropriados de recuperação, resulta em supercompensação metabólica ao treinamento, refletida como aumento na capacidade aeróbica e/ou força muscular. Visto como contínuo, os processos de intensificação do treinamento e o estresse relacionado à supercompensação, o aumento da sobrecarga ou do estresse poderá, em algum momento, acarretar a quebra da homeostase e a queda temporária da função (supra-alcance - OR ou supra-alcance funcional - FOR). Quando a sobrecarga excessiva de treinamento é combinada com recuperação inadequada há instalação do estado de supratreinamento (OT) ou supra-alcance não funcional (NFOR). O OT excede o OR, cujo pico é também o limiar do OT resultando em desadaptações fisiológicas e queda crônica do desempenho físico. A forma crônica de desadaptação fisiológica ao treinamento físico é chamada de síndrome do supertreinamento (OTS). A própria expressão da síndrome denota a etiologia multifatorial do estado e reconhece que o exercício não é necessariamente seu único fator causal. O diagnóstico de OTS é baseado na recuperação ou não do desempenho. Não há biomarcador objetivo para OTS. A distinção entre OTS e NFOR (supratreinamento extremo) é dependente de desfecho clínico e exclusão diagnóstica de doenças orgânicas, mais comuns na OTS. Também a diferença entre OR e OT é sutil e nenhum de seus marcadores bioquímicos pode ser universalizado. Não há evidências confirmatórias que OR evolui para OT ou que os sintomas de OT são piores dos que os de OR. Apenas pela fadiga aguda e queda de rendimento experimentada em sessões isoladas de treinamento, não é possível diferenciar presentemente os estados de OR e OT. Isto é devido, parcialmente, à variabilidade das respostas individuais ao treinamento e à falta de ambos instrumentos diagnósticos e estudos bem controlados.

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Several studies have demonstrated that caffeine improves endurance exercise performance but the mechanisms are not fully understood. Possibilities include increased free fatty acid (FFA) oxidation with consequent sparing of muscle glycogen as well as enhancement of neuromuscular function during exercise. The present study was designed to investigate the effects of caffeine on liver and muscle glycogen of 3-month old, male Wistar rats (250-300 g) exercising by swimming. Caffeine (5 mg/kg) dissolved in saline (CAF) or 0.9% sodium chloride (SAL) was administered by oral intubation (1 mu l/g) to fed rats 60 min before exercise. The rats (N = and-IO per group) swam bearing a load corresponding to 5% body weight for 30 or 60 min. FFA levels were significantly elevated to 0.475 +/- 0.10 mEq/l in CAF compared to 0.369 +/- 0.06 mEq/l in SAL rats at the beginning of exercise. During exercise, a significant difference in FFA levels between CAF and SAL rats was observed at 30 min (0.325 +/- 0.06 vs 0.274 +/- 0.05 mEq/l) but not at 60 min (0.424 +/- 0.13 vs 0.385 +/- 0.10 mEq/l). Blood glucose showed an increase due to caffeine only at the end of exercise (CAF = 142.1 +/- 27.4 and SAL = 120.2 +/- 12.9 mg/100 ml). No significant difference in liver or muscle glycogen was observed in CAF as compared to SAL rats, at rest or during exercise. Caffeine increased blood lactate only at the beginning of exercise (CAF = 2.13 +/- 0.2 and SAL = 1.78 +/- 0.2 mmol/l). These data indicate that caffeine (5 mg/kg) has no glycogen-sparing effect on rats exercising by swimming even though the FFA levels of CAF rats were significantly higher at the beginning of exercise.

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Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)