166 resultados para glucose

Interaction between insulin (VNTR) and hepatic lipase (LIPC-514c>T) variants on the response to an oral glucose tolerance test in the EARSII group of young healthy men

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The glucose response to an oral fat tolerance test in young men with a paternal history of premature myocardial infarction: possible early indication of insulin resistance. The EARS 2 Study

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Low and conventional dose cyclopenthiazide on glucose and lipid metabolism in mild hypertension.

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Direct and indirect effects of the anorectic hormone obestatin on glucose regulation and insulin secretion in mice

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Characterisation of novel glucose-dependent insulinotropic polypeptide (GIP) antagonists generated through amino acid substitution at position Glu(3)

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Daily administration of GIP receptor antagonist alleviates glucose intolerance and counters insulin resistance in an animal model of obesity and Type 2 diabetes

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N-terminally modified analogues of glucose-dependent insulinotropic polypeptide exhibit improved insulinotropic activity and antihyperglycaemic potential compared with glucagon-like peptide-1 counterparts

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Comparative studies with exendin(9-39)amide and the novel (Pro(3))GIP receptor antagonist indicates that glucose-dependent insulinotropic polypeptide is the major hormone of the enteroinsular axis

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Non-Enzymatic Electrochemical Glucose Detection

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Second messenger pathways and transcription factors involved in PDGF-beta receptor upregulation in raised glucose conditions.

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The effect of high glucose and cyclic stress on pathways dependent on extracellular-regulated kinase (ERK) activation in vascular smooth muscle cells.

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Elevated glucose concentration potentiates vascular smooth muscle cell chernotaxis via phosphoinositide 3-kinase-p110 beta, protein kinase B and protein kinase C beta II

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High glucose mediates vascular smooth muscle cell survival through the activation of protein kinase B and extracellular regulated kinase 1/2.

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Vascular smooth muscle cell chemotaxis is potentiated by glucose via phosphoinositide 3-kinase-p110 beta, Akt, ERK1/2 and protein kinase C beta 11 dependent pathways.

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The involvement of PI3K and MAPK in regulation of PDGF-beta receptor number by glucose

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